146 cell culture 147 immortalized mouse renal tubular epithelial cells rtecs Search Results


146 cell culture 147 immortalized mouse renal tubular epithelial cells rtecs  (ATCC)
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ATCC 146 cell culture 147 immortalized mouse renal tubular epithelial cells rtecs
146 Cell Culture 147 Immortalized Mouse Renal Tubular Epithelial Cells Rtecs, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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renal tubular epithelial cell rtec  (ATCC)
99
ATCC renal tubular epithelial cell rtec
Renal Tubular Epithelial Cell Rtec, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/146+cell+culture+147+immortalized+mouse+renal+tubular+epithelial+cells+rtecs/Renal+Epithelial+Cell+Basal+Medium/ppr0320154-38-3-11
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renal tubular epithelial cell rtec - by Bioz Stars, 2026-09
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tubular epithelial cells rtecs  (ATCC)
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ATCC tubular epithelial cells rtecs
Tubular Epithelial Cells Rtecs, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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rat renal tubule epithelial cells  (ATCC)
96
ATCC rat renal tubule epithelial cells
Trps1 promotes the proliferation of renal tubular <t>epithelial</t> cells after H/R. Renal tubular epithelial cells <t>(NRK-52E)</t> were subjected to hypoxic conditions for 4 hours followed by reoxygenation (H/R model). (A) After H/R, the PCNA and trps1 levels of NRK-52E cells were detected by Western blotting, and (B and C) the relative levels to β-actin were quantified (*P<0.05 versus control). (D) The immunohistochemical analyses for Trps1 and PCNA coexpression in NRK-52E cells were performed at 6 and 24 hours after H/R injury. (E) The PCNA protein levels were increased in Trps1-overexpressed NRK-52E cells at 24 hours after H/R, and (F) its relative levels to β-actin were quantified. (G) The PCNA protein levels were reduced in Trps1 knockdown NRK-52E cells at 24 hours after H/R, and (H) its relative levels to β-actin were quantified (*P<0.05 versus 0h).
Rat Renal Tubule Epithelial Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/146+cell+culture+147+immortalized+mouse+renal+tubular+epithelial+cells+rtecs/NRK-52E/pmc05280010-287-1-11
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rat renal tubule epithelial cells - by Bioz Stars, 2026-09
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human renal tubular epithelial cells rtecs hk 2  (ATCC)
99
ATCC human renal tubular epithelial cells rtecs hk 2
Trps1 promotes the proliferation of renal tubular <t>epithelial</t> cells after H/R. Renal tubular epithelial cells <t>(NRK-52E)</t> were subjected to hypoxic conditions for 4 hours followed by reoxygenation (H/R model). (A) After H/R, the PCNA and trps1 levels of NRK-52E cells were detected by Western blotting, and (B and C) the relative levels to β-actin were quantified (*P<0.05 versus control). (D) The immunohistochemical analyses for Trps1 and PCNA coexpression in NRK-52E cells were performed at 6 and 24 hours after H/R injury. (E) The PCNA protein levels were increased in Trps1-overexpressed NRK-52E cells at 24 hours after H/R, and (F) its relative levels to β-actin were quantified. (G) The PCNA protein levels were reduced in Trps1 knockdown NRK-52E cells at 24 hours after H/R, and (H) its relative levels to β-actin were quantified (*P<0.05 versus 0h).
Human Renal Tubular Epithelial Cells Rtecs Hk 2, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/146+cell+culture+147+immortalized+mouse+renal+tubular+epithelial+cells+rtecs/HK-2/10__1096_slash_fj__202402443r-21-0-13
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human renal tubular epithelial cells rtecs hk 2 - by Bioz Stars, 2026-09
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mouse renal tubule epithelial cells mtecs  (ATCC)
96
ATCC mouse renal tubule epithelial cells mtecs
Trps1 promotes the proliferation of renal tubular <t>epithelial</t> cells after H/R. Renal tubular epithelial cells <t>(NRK-52E)</t> were subjected to hypoxic conditions for 4 hours followed by reoxygenation (H/R model). (A) After H/R, the PCNA and trps1 levels of NRK-52E cells were detected by Western blotting, and (B and C) the relative levels to β-actin were quantified (*P<0.05 versus control). (D) The immunohistochemical analyses for Trps1 and PCNA coexpression in NRK-52E cells were performed at 6 and 24 hours after H/R injury. (E) The PCNA protein levels were increased in Trps1-overexpressed NRK-52E cells at 24 hours after H/R, and (F) its relative levels to β-actin were quantified. (G) The PCNA protein levels were reduced in Trps1 knockdown NRK-52E cells at 24 hours after H/R, and (H) its relative levels to β-actin were quantified (*P<0.05 versus 0h).
Mouse Renal Tubule Epithelial Cells Mtecs, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/146+cell+culture+147+immortalized+mouse+renal+tubular+epithelial+cells+rtecs/TCMK-1/pm37248964-51-11-24
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mouse renal tubule epithelial cells mtecs - by Bioz Stars, 2026-09
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Image Search Results


Trps1 promotes the proliferation of renal tubular epithelial cells after H/R. Renal tubular epithelial cells (NRK-52E) were subjected to hypoxic conditions for 4 hours followed by reoxygenation (H/R model). (A) After H/R, the PCNA and trps1 levels of NRK-52E cells were detected by Western blotting, and (B and C) the relative levels to β-actin were quantified (*P<0.05 versus control). (D) The immunohistochemical analyses for Trps1 and PCNA coexpression in NRK-52E cells were performed at 6 and 24 hours after H/R injury. (E) The PCNA protein levels were increased in Trps1-overexpressed NRK-52E cells at 24 hours after H/R, and (F) its relative levels to β-actin were quantified. (G) The PCNA protein levels were reduced in Trps1 knockdown NRK-52E cells at 24 hours after H/R, and (H) its relative levels to β-actin were quantified (*P<0.05 versus 0h).

Journal: Journal of the American Society of Nephrology : JASN

Article Title: Transcription Factor Trps1 Promotes Tubular Cell Proliferation after Ischemia-Reperfusion Injury through cAMP–Specific 3′,5′-Cyclic Phosphodiesterase 4D and AKT

doi: 10.1681/ASN.2016010009

Figure Lengend Snippet: Trps1 promotes the proliferation of renal tubular epithelial cells after H/R. Renal tubular epithelial cells (NRK-52E) were subjected to hypoxic conditions for 4 hours followed by reoxygenation (H/R model). (A) After H/R, the PCNA and trps1 levels of NRK-52E cells were detected by Western blotting, and (B and C) the relative levels to β-actin were quantified (*P<0.05 versus control). (D) The immunohistochemical analyses for Trps1 and PCNA coexpression in NRK-52E cells were performed at 6 and 24 hours after H/R injury. (E) The PCNA protein levels were increased in Trps1-overexpressed NRK-52E cells at 24 hours after H/R, and (F) its relative levels to β-actin were quantified. (G) The PCNA protein levels were reduced in Trps1 knockdown NRK-52E cells at 24 hours after H/R, and (H) its relative levels to β-actin were quantified (*P<0.05 versus 0h).

Article Snippet: Reagents Rat renal tubule epithelial cells (NRK-52E) were purchased from the American Type Culture Collection (Manassas, VA).

Techniques: Western Blot, Control, Immunohistochemical staining, Knockdown